WEBVTT

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Okay, so let's do a CRAM
session for MSK upper extremity. As always,

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I'm going to stick to the need
to know stuff. I'm not going

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to bother your time with the stuff
that I really feel is very low yield

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and that you don't need to know. So let's go ahead and get started.

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As always, thank you so much
for the really nice comments. You

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guys are just the nicest people,
So thank you so much. Let's go

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ahead and start with the shoulder and
they'll work our way down. So anterior

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gleno humeral dislocation, so this is
your most common type. Anterior dislocation is

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going to account for around ninety five
to ninety seven percent of cases of shoulder

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dislocations. Now, as far as
the mechanism of injury, it's going to

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be a blow to the abducted,
externally rotated and extended arm. So that's

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the most common mechanism of injury.
So think like blocking a basketball shot your

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arms up and out. Less commonly
is going to be a blow to the

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posterior humorous or a fault on an
outstretched arm can also cause an anterior dislocation,

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but focus on that arm being abducted. Abducted away from the body,

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extended and externally rotated, and then
something hitting the arm in that position.

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Non physical exam. You need to
know that you're looking for an abducted,

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abducted externally rotated arm, So an
anterior dislocation of the shoulder causing the arm

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to be slightly abducted and externally rotated. Diagnosis, you're going to do an

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X ray. X ray of the
shoulder is normally going to be enough to

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diagnose. Going to get an ap
view, scapular y view, axillary view.

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A few associated injuries you should know
with an anterior dislocation. There's the

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hill Sacks, bank art lesions,
axillary nerve injury. It wouldn't go crazy

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memorizing the specific injuries what they involved. Just know they're associated with anterior dislocation

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hill sacks real quick. It's a
cortical depression. Hill sax lesion is a

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cortical depression in the humoral head made
by the glenoid rim. Bank art legions

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occur when the glenoid labrim is disrupted
during the dislocation, and a bone fragment

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is a vols and an axillary nerve
injury. This one's probably the most important

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associated injury to know. So it's
really important to assess the nerve vascular status

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in patients with an anterior shoulder dislocation
because axillary nerve disfunction can actually show up

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to up to forty two percent of
patients with an anterior dislocation. So you're

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gonna look for a loss of sensation
on what's called the shoulder bags distribution.

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It's basically like on a cop uniform
or a military uniform. Just think of

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the area of the arm that the
shoulder badge would be. Where they have

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a badge on the arm. That's
where the axillary nerve innervates, and that's

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where they may have this loss of
sensation. They may also have deltoid muscle

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weakness. So just be familiar that
axillary nerve dysfunction is common in anterior dislocations.

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As far as treatment reduced in sling, there's not much to know here,

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certainly nothing to memorize. You reduce
the dislocation, mobilize it with the

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sling, and then assess for axillary
nerve dysfunction pre and post reduction. Okay,

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so there's a lot of random things
to know for anterior dislocations. So

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the way that you're going to remember
them is by remembering a guy named Antonio.

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So this is visualization times is much
easier on the YouTube channel. I

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have a nice little picture I'm made
in MS paint, but let's just try

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to visualize this. So Antonio is
this guy and in one hand he's holding

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a picture and in the other hand
he's holding an axe. The picture that

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he's holding is a bank on top
of a hill like this nice pretty hill

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with a sunset in the background.
And then there's a bank sitting on top

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of the hill. That's the picture. The other hand he's holding an axe.

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Now he's holding both of these up
and out by his sides. His

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arms are abducted and they're externally rotated. So Antonio helps you to remember this

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as an anterior dislocation. Antonio anterior
dislocation. The picture that he's holding with

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the bank on top of a hill
helps you remember bank art lesions and hill

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sack lesions that can commonly be seen
in anterior dislocations. And then the axe

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he's holding in his other hands helps
you remember axillary nerve dysfunction that's the most

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common in anterior dislocations. And then
finally, the position of his arms.

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Like I said, they're abducted and
externally rotated. That helps you remember both

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the way the arms usually positioned during
the physical exam and during the injury too.

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So remember a guy named Antonio holding
a picture of a bank on top

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of a hill on one hand holding
an axe, and the other both arms

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are abducted and externally rotated. And
that should be all you need to know

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for anterior dislocations for the exam,
moving onto posterior glenohumeral dislocation. So these

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are much less common compared to anterior
They only occur in around two to four

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percent of patients. Mechanism of injury
for anterior dislocations wasn't very high yield,

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but for posterior dislocations it is,
and it's because it's unique. And if

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you listen to any of my other
content, you always know anytime there's something

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unique, they're going to ask you
questions about it, So keep that in

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mind. Mechanism for posterior dislocations is
going to be seizure and electric shock,

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so those are the ones that you
need to know. Of course, trauma

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like a blow to the anterior portion
of the shoulder can cause a posterior dislocation,

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But what you should focus on is
violent muscle contractions following a seizure or

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electrocution, because those are the unique
components that they like to ask questions about

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seizure or electric shock. Remember those
for your mechanism of injury in a posterior

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dislocation. Now physical exam opposite of
anterior So for a posterior dislocation, it's

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going to be adducted and internally rotated. So usually the patient's going to hold

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their arm in adduction and internal rotation
and is usually unable to externally rotate again.

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And that's different than anterior dislocations,
which was abducted, abducted and externally

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rotated. Now, diagnosis, you're
going to get an X ray, so

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you'll do your normal routine shoulder views, apyve you, etc. But be

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aware of the light bulb sign on
posterior dislocations because they may ask about this.

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So on the AP view, the
humoral head is going to be internally

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rotated and because of this, the
twoberocities of the humorous they're not going to

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be visible. They're going to be
normally, they're project out laterally, but

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they're not going to be visible because
the humoral heads internally rotated. And because

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of this, I have to take
my word for it, the humoral head

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kind of takes on the circular appearance
and supposedly looks like a light bulb.

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I guess it kind of does.
So anyways, if you hear a light

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bulb sign, be thinking. Posterior
dislocation treatment is going to be the same

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as an interior You're going to reduce
and sling. Okay, So for posterior

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dislocation, there's a few things that
you need to know. Remember your caesar

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seizure and shock can be the mechanism
of action, mechanism of injury. Remember

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the arm is likely going to be
adducted and internally rotated. And remember the

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light bulb sign. Those are the
high yel things. And the way that

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you remember that is I want you
to visualize this warning on a poster board.

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So on the posterboard there's going to
be a picture, and on the

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posterboard there's going to be a picture
of a broken light bulb. There's going

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to be a finger, and then
there's going to be a picture of a

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guy being shocked. And it says
if you add your finger into a broken

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light bulb, you'll get shocked,
So add an into if you add your

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finger into, that helps you remember
adducted and internal rotation. So if you

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add your finger into adducted internal rotation, remember that's the most common presentation,

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the broken light bulb, because you
remember the light bulb sign on X ray

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scene of posterior dislocations and shocked because
remember the unique mechanism of injury, electric

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shock, and seizure. And then
again this is all on a posterboard because

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that posterboard helps remember posterior dislocation.
So again, remember a poster board,

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and on the poster board it says
if you add your finger into a broken

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light bulb, you'll get shocked.
Visual a posterboard with a finger a plus

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sign, a broken light bulb and
a guy being shocked, and you'll remember

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everything you need to know for posterior
dislocations. All right, moving on to

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a rotator cuff injury. This is
definitely a high heel topic. There's a

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good deal to know for this,
and you'll likely get a question on this.

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So let's go over a rotator cuff
tears and impingement. So first,

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you need to know your muscles in
the rotator cuff. I'm sure you've all

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heard of the famous mnemonic sits S
I S so superspinatus infraspinatus tarifs minor and

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subscapularis mechanism of injury for rotator cuff. Injury is going to be a few

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different things. So degeneration like chronic
degenerative tear that's usually going to be seen

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in older patients. Impingement. Chronic
and pingement can also lead to a tear

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and then overload, so like tension
overload and athletes are continually throwing like in

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baseball or jobs with repetitive overhead movement. In general, the cause of rotator

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cuff tears, it's multifactorial degeneration and
pingement, over they can all contribute.

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There's not really anything I'd say is
super important to memorize. The high yield

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things for a rotator cuff is going
to be the presentation of physical exam tests,

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so clinical manifestations. There's two things
to know here. Overhead pain and

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pain at night. That's the key. So pain exacerbated by overhead activities.

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So the vignett they're going to describe
the patient that's maybe reaching up to the

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shelf and experiencing pain, brushing their
hair and having pain things like that,

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definitely look out for. The location
of the pain is usually going to be

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over their lateral deltoid, so the
anterior lateral portion of the shoulder. And

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then they also may describe pain at
night, so complaining like when the patient's

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lying on the shoulder at night,
they're experiencing pain. That's the classic presentation.

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Will look out for in vignettes the
pain over their lateral deltoid when they're

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reaching overhead, or complaining of pain
at night and real life, though it's

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possible that they may just develop weakness
decreased range of motion. Other studies even

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suggest a large portion of rotator cuff
tears. They're actually asymptomatic. But for

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the exam pain with overact, pain
with overhead activities, pain at night done

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physical exam. So there's a lot
of special maneuvers for the rotator cuff when

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you're looking for impingement and tears.
So three that I feel you should be

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familiar with at seem to be tested
on the most is the Hawkins test,

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the near tests, and the empty
Can test. Again, these are for

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both impingement and tears. So let's
start with the Hawkin and the Near test.

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These are both really good tests to
test to assess for impingement. So

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the Hawkins test, also known as
the Hawkins Kennedy impingement test, the patient's

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going to have their shoulders stabilize and
positioned in ninety degrees of elevation. The

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elbow is going to be bent to
ninety degrees. The examiner is going to

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place internal force on the patient's shoulder, and then any reproduction of pain elicited

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by the internal rotation would be a
positive test for impingement. Again, if

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you need visuals on any of these, check out the YouTube channel. Have

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a lot of different pictures and things
like that that kind of help. So

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the way that I used to remember
this test, the Hawkins test, was

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if you can visualize when you were
a kid, did you ever call someone

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a chicken? You do the little
arm flapping thing like wings. That's exactly

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what this looks like when you're doing
the Hawkins test. Their shoulders up,

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their elbows bent. So when I
see Hawkins test, it makes me think

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of a hawk flapping its wings.
And I'll help you remember this is the

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one where it looks like you're flapping
your wings like a bird or a hawk,

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with the elbow bent, shoulder elevator
elevated, and the examiner applying internal

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rotation. That's exactly what this looks
like. You'll have to take my word

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for it or check out some pictures
of the Hawkins test, but it looks

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like somebody like flapping their wings like
a hawk. So again remember that in

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your os keys and things like that, you'll remember what the Hawkins test looks

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like and what it involves. So
remember Hawkins tests flapping your wing like a

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hawk. I let's talk about the
near test. So the near test,

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the shoulder is going to be forcibly
flexed and internally rotated. So one hand

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is going to be placed on the
shoulder and the scapular area to keep the

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shoulder from shrugging. The other hand
is going to passively lift the arm all

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the way up to the ear.
The arm is going to be internally rotated

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with the thumb facing down, and
then any reproduction of pain is a positive

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test for impingement. May see some
variations of this test with the arm and

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neutral position rather than internal, but
most of the time it's going to have

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internal rotation. Ice Remember near is
by the ear because and this test,

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your arm goes all the way up
and the shoulder winds up right next to

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the ear. So when you see
the near test, remember near is the

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one by the ear. And then
finally, the empty can test, also

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known as the Job's test. This
is a good test to evaluate superspinatus function.

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Sometimes it's considered the gold standard test
due to how well it isolates the

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superspinatus in the way that the arm
is position to really isolates it well.

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So the patient's going to place a
straight arm out at about ninety degrees of

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ab duction and thirty degrees of forward
flection, then internally rotating the arm completely.

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The thumb is going to be pointing
down. And then the patient then

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resists. The clinicians attempts to depress
the arm, so you're trying to push

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it down. They're resisting. So
again the patient raises their arm forward,

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elbow extended, thumbs down as if
emptying a can. Examiner's going to apply

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pressure to the top of the arm, pushing it down. The patient attempts

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to resist. This. Pain without
weakness is going to suggest tendinopathy. Pain

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with weakness is consistent with the tendon
tear There's a lot of other tests,

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but in my experience, those are
the three that seem to be most often

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tested on, so definitely be familiar
with those three. Now, treatment conservative

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versus surgical repair, there's nothing really
specific to know here. Treatment is all

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going to depend on. It's really
a several factors like the duration of the

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symptoms, the type of tear partial
versus full thickness, patient age, activity

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level, etc. That's going to
guide the treatment as to whether or not

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they're going to need surgical repair or
if they're better suited for say physical therapy.

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Now, if we're talking about tendonitis
and the impingement of the rotator cuff,

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treatment is primarily going to be physical
therapy and sets, etc. You're

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gonna find with most of MSK treatment
is pretty repetitive. It's not very high

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yield. That's why I kind of
run through them quickly because there's just not

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much to know from an exam standpoint, So from breezing over and I'm skipping

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in some cases, it's just because
there's nothing really for you to memorize.

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If there's something high yield for treatment, I'll definitely make sure to bring it

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to your attention, all right,
so let's move on another area of the

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shoulder known as adhesive capsulitis, also
known as a frozen shoulder. So this

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00:13:03.039 --> 00:13:09.279
is a painful and stiff glenohumeral joint
that has lost its distensibility and range of

203
00:13:09.320 --> 00:13:11.159
motion. So it's exactly what it
sounds like. It's a frozen shoulder.

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00:13:11.159 --> 00:13:13.960
It's hard to move. Passive and
active range of motion are going to be

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00:13:15.000 --> 00:13:18.519
substantially reduced. Risk factors diabetes,
this is a big one. Remember that

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00:13:18.559 --> 00:13:22.480
frozen shoulder is very common in diabetics, and the vignette it will very very

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00:13:22.480 --> 00:13:26.120
likely be a diabetic patient. There's
actually a study that showed the incidence of

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00:13:26.200 --> 00:13:31.600
frozen shoulder and patients with long standing
type one diabetes had a lifetime prevalence of

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00:13:31.679 --> 00:13:35.039
up to seventy six percent. So
diabetic patients remember that the other risk factor

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00:13:35.080 --> 00:13:39.960
to know is thyroid disorders, particularly
hypothyroidism, may increase the risk for frozen

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00:13:41.000 --> 00:13:43.480
shoulder as much as two point seven
times. And then there's some other causes

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00:13:43.480 --> 00:13:48.360
that you shouldn't memorize should not memorize, like dyslipidemia, prolonged immobilization, stroke,

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00:13:48.399 --> 00:13:52.440
auto immune disease. Secondary problem after
shoulder injury. Focus on diabetes,

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00:13:52.519 --> 00:13:56.960
they got that sticky shoulder from all
that extra sugar and thyroid disorders. And

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00:13:58.000 --> 00:14:01.440
then also know that this is very
common in women, particularly in the fifth

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00:14:01.480 --> 00:14:05.159
and sixth decades of life. It's
actually really rare to see the impatience under

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00:14:05.200 --> 00:14:09.039
forty. So in the vignette,
be looking for a female in their fifties

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00:14:09.120 --> 00:14:13.320
or sixties with diabetes or thyroid disorder. For diagnosis, There's nothing I really

219
00:14:13.360 --> 00:14:16.519
know here. Frozen shoulder is very
much a clinical diagnosis made on the basis

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00:14:16.559 --> 00:14:22.720
of the medical history. The physical
exam imaging really has very little value except

221
00:14:22.720 --> 00:14:26.159
just to rule out your differentials like
osteoarthritis and things like that. Treatment is

222
00:14:26.200 --> 00:14:31.519
also pretty low yield most cases.
Frozen shoulders is self limited condition. Physical

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00:14:31.559 --> 00:14:35.480
therapy is the most commonly employed treatment
option. Google corticoid injections and SAIDs other

224
00:14:35.519 --> 00:14:39.240
supportive measures, but nothing really to
commit to memory here, all right,

225
00:14:39.279 --> 00:14:45.840
So moving on down the arm.
Supercondular humorous fractures, so supercondular fractures of

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00:14:45.879 --> 00:14:50.320
the distal humorous You're going to see
this most common in children two to seven

227
00:14:50.399 --> 00:14:54.639
years old, so most frequently in
children two to seven years old. So

228
00:14:54.799 --> 00:14:58.360
be looking for a child in the
vignette. Supercondular fractures are actually the most

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00:14:58.399 --> 00:15:03.600
common mediatric elbow fracture as far as
the mechanism of injury. Foosh with the

230
00:15:03.639 --> 00:15:09.080
elbow extended. So foosh foosh with
the elbow hyper extended. So you're gonna

231
00:15:09.080 --> 00:15:13.720
hear me saying that a lot.
So extension fractures are actually going to account

232
00:15:13.720 --> 00:15:18.519
for approximately ninety five percent of all
supercondular fractures, and this is typically going

233
00:15:18.519 --> 00:15:22.200
to be from a foosh mechanism.
So foosh stands for fall on outstretched hand

234
00:15:22.399 --> 00:15:24.840
and then in this case, the
elbow is going to be hyper extended.

235
00:15:26.039 --> 00:15:31.480
Like we talked about before, this
is really seen very commonly in children,

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00:15:31.480 --> 00:15:35.159
and the reason is because children have
this increased flexibility in their joints, They

237
00:15:35.159 --> 00:15:39.600
have this ligamentous laxity, and when
they fall with their arm extended out,

238
00:15:39.919 --> 00:15:43.600
they're more susceptible to hyper extending the
arm when they fall, and then hyper

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00:15:43.600 --> 00:15:48.600
extension is what generally leads to this
type of fracture, to a supercondular fracture.

240
00:15:48.639 --> 00:15:52.600
So remember supercondular foosh with hyper extension
diagnosis, you're going to get an

241
00:15:52.639 --> 00:15:56.879
X ray and once you're looking for
here is fat pad. So the only

242
00:15:56.960 --> 00:15:58.919
thing I really think you should be
aware of, at least for imaging when

243
00:16:00.039 --> 00:16:03.840
comes to supercondular fractures for the exam
are fat pads. So in a non

244
00:16:03.919 --> 00:16:08.200
displaced or minimally displaced fracture of this
region, sometimes you actually can't see the

245
00:16:08.200 --> 00:16:11.919
fracture. Sometimes it can't be visualized. And the only indication that a fracture

246
00:16:12.000 --> 00:16:17.799
is present is an elbow effusion like
seen in an anterior sale or posterior fat

247
00:16:17.840 --> 00:16:21.360
pads signs. So let's talk about
fat pads for a second. In the

248
00:16:21.399 --> 00:16:23.279
elbow, you have fat pads.
Usually they're not visible on X ray.

249
00:16:23.519 --> 00:16:26.559
I'm going to talk about this a
little bit more when we get to radio

250
00:16:26.600 --> 00:16:30.200
head fractures. Next your interior verus
posterior which you can be normal, which

251
00:16:30.200 --> 00:16:36.159
you cannot. But generally fat pads, particularly posterior fat pads, they're not

252
00:16:36.279 --> 00:16:40.320
visible unless you have an acute injury
and then this blood starts to collect.

253
00:16:40.559 --> 00:16:44.840
This hemarthrosis elevates the fat pads out
of the coronoid and the electronoid electro non

254
00:16:44.879 --> 00:16:48.919
fossa, and that's what makes them
visible on radiographs and actually in more than

255
00:16:48.039 --> 00:16:52.919
ninety percent of cases where imaging shows
posterior fat pad displacement, a fracture is

256
00:16:52.919 --> 00:16:56.720
seen on initial or follow up radiographs. So at the elbow will be looking

257
00:16:56.720 --> 00:17:02.519
for your fat pads and then also
be aware there is something called an interior

258
00:17:02.679 --> 00:17:06.799
humeral line. If there's any displacement
of this line, this can also indicate

259
00:17:06.799 --> 00:17:08.519
a displaced fracture. I don't think
they're going to ask you about this though,

260
00:17:08.680 --> 00:17:12.559
Just really focus on your fat pads
now. Complications. There's a lot

261
00:17:12.559 --> 00:17:18.680
of complications that can arise from a
supercondular fracture, vascular insufficiency, nerve injury,

262
00:17:18.720 --> 00:17:22.240
compartment syndrome, So I want to
make sure you're doing a complete neurovascular

263
00:17:22.240 --> 00:17:25.079
evail in these patients. You need
to check the sensory and the motor function

264
00:17:25.359 --> 00:17:27.400
of the medial, of the median, radio owner nerves. You want to

265
00:17:27.400 --> 00:17:30.839
assess the radio and brakual pulses.
What you really need to be looking out

266
00:17:30.839 --> 00:17:34.799
for. An exam question when it
comes to complications of a supercondular fracture is

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00:17:34.839 --> 00:17:41.920
something called Volkman's schemic contracture. So
this isn't common, but it's definitely tested

268
00:17:41.920 --> 00:17:44.440
on. So when you have this
type of fracture and you have a vascular

269
00:17:44.480 --> 00:17:48.680
injury and swelling. This can potentially
lead to compartment syndrome, and if that

270
00:17:48.759 --> 00:17:52.319
compartment syndrome isn't treated in a timely
manner, the associated a schemia and infraction

271
00:17:52.359 --> 00:17:56.920
can lead to Volkman's a schemic contraction. So what this causes is you have

272
00:17:57.000 --> 00:18:02.240
this fixed flection of the elbow,
the extension of the MCP joints. But

273
00:18:02.359 --> 00:18:06.160
it really simply it's this claw like
deformity of the hands fingers and the risk

274
00:18:06.640 --> 00:18:10.440
risk. So for the exam,
if you see Volkman's contracture right away,

275
00:18:10.480 --> 00:18:14.039
be thinking of a supercondular fracture,
because while this can be seen with other

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00:18:14.039 --> 00:18:18.000
types of injuries of the arm,
it's most commonly seen with supercondular fractures.

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00:18:18.000 --> 00:18:23.160
So associate those two in your head
Volkman's supercondular, supercondular Volkman's or just remember

278
00:18:23.200 --> 00:18:27.680
what I did and remember my vocal
at my Volkswagens in super condition, so

279
00:18:27.839 --> 00:18:37.480
Volkswagen helps remember Volksman contracture. Supercondition
helps remember supercondular fracture. So my Volkswagen's

280
00:18:37.519 --> 00:18:45.799
in supercondition Volkswagen Volkman's contracture supercondition helps
you remember supercondular fracture. All right,

281
00:18:45.880 --> 00:18:51.359
so treatment is going to be splint
verse surgical most displaced or minimally displaced fractures,

282
00:18:51.359 --> 00:18:55.319
you're going to apply a posterior your
splint and sling. Displaced fractures generally

283
00:18:55.519 --> 00:19:00.119
be repaired surgically closed versus open reduction
with percutaneous pin play. Nothing really to

284
00:19:00.160 --> 00:19:04.440
memorize here. So for supercondular fractures, remember this is very likely going to

285
00:19:04.440 --> 00:19:07.880
be a young child in the vignette. It's going to be a foosh injury

286
00:19:07.880 --> 00:19:11.759
with hyper extension. You remember fall
on outstretched hand, Look for your fat

287
00:19:11.799 --> 00:19:17.279
pads on X ray, and then
finally remember your Volksman's contracture for potential complications.

288
00:19:17.279 --> 00:19:22.359
Remember by Volkswagens in supercondition. Okay, so next radiohead fractures. There's

289
00:19:22.359 --> 00:19:25.319
really just a few things to know
here. The mechanism of injury is going

290
00:19:25.359 --> 00:19:29.960
to be another foosh, So another
foushure like in supercondular humeral fractures and many

291
00:19:29.960 --> 00:19:33.640
other fractures of the arm. Really, so falling onto an outstretched hand is

292
00:19:33.680 --> 00:19:37.200
the most common mechanism for radiohead or
neck fractures. Because this type of injury

293
00:19:37.480 --> 00:19:44.079
there's this sudden impaction on the radius
and onto the capitulum. So as far

294
00:19:44.079 --> 00:19:45.559
as diagnosis, you're going to get
your X ray. You're looking for fat

295
00:19:45.559 --> 00:19:49.519
pads here again. Now, the
fat pads in radiohead fractures are even more

296
00:19:49.559 --> 00:19:56.039
important than supercondular fractures that we just
discussed before, because non displaced radiohead fractures

297
00:19:56.039 --> 00:20:00.880
can very very often be missed and
an elevated interior and posterior fat pads maybe

298
00:20:00.960 --> 00:20:07.160
the only sign of an occult radiohead
fracture. So be looking for those fat

299
00:20:07.200 --> 00:20:08.720
pads in the vignette and in real
life. Wanted to give you a little

300
00:20:08.720 --> 00:20:12.680
bit of extra knowledge while we're on
the topic of fat pads, so I

301
00:20:12.680 --> 00:20:18.039
interior fat pads can actually be a
normal variant, particularly in children. While

302
00:20:18.039 --> 00:20:23.119
a posterior fat pad should really never
be visible. It'll almost always indicate trauma.

303
00:20:23.240 --> 00:20:26.519
So again, remember anterior fat pads
can sometimes be a normal finding,

304
00:20:26.720 --> 00:20:32.200
while posterior will almost always indicate a
fusion caused by trauma. And I used

305
00:20:32.200 --> 00:20:36.119
to remember and tieror fat pads and
tior with an A can be AOKA,

306
00:20:36.640 --> 00:20:41.720
and posterior fat pads with a P
are always problematic. So anterior AOKA posterior

307
00:20:41.720 --> 00:20:45.240
problematic helps you remember which one can
be normal, which one is always bad.

308
00:20:45.559 --> 00:20:48.359
Treatment is going to be splint for
a surgical nothing really to know here.

309
00:20:48.480 --> 00:20:51.920
Depending on the severity of the fracture
and mobilization with a sling or posterior

310
00:20:51.960 --> 00:20:56.119
splint may be sufficient for surgical repair
for your displaced For more severe fractures,

311
00:20:56.440 --> 00:21:00.640
again, little mill for radiohead fractures, just remember your food injury. You

312
00:21:00.720 --> 00:21:04.039
know your fat pad sign for those
occult fractures. Occult fractures. If I

313
00:21:04.039 --> 00:21:07.440
didn't mention this before, it just
means a hidden fracture that's not visible on

314
00:21:07.519 --> 00:21:12.759
imaging. Let's move on to ulner
shaft or night stick fractures. Really,

315
00:21:12.880 --> 00:21:15.559
very little to know for this one. I only bring it up for some

316
00:21:15.599 --> 00:21:18.599
reason. This does come up on
exams. Main takeaway here is to know

317
00:21:18.640 --> 00:21:22.039
the type of injury that's related to
the specific fracture. So this is going

318
00:21:22.079 --> 00:21:26.400
to be an isolated ulner shaft fracture. The mechanism of injury is going to

319
00:21:26.440 --> 00:21:30.319
be a direct blow to the forearm. So these types of fractures almost always

320
00:21:30.400 --> 00:21:33.559
result from a direct blow to the
forum. So this happens when the patient

321
00:21:33.640 --> 00:21:37.079
was using the forum to protect their
head from their torso, like from a

322
00:21:37.119 --> 00:21:41.079
blow with a night stick or a
pool stick or something like that. So

323
00:21:41.119 --> 00:21:44.319
if you're familiar with the mechanism of
injury, when you see it in a

324
00:21:44.400 --> 00:21:47.599
vignette, you'll know you'll be looking
for an ulner fracture. In the answers,

325
00:21:47.920 --> 00:21:52.079
treatment is going to be non operative
treatment verse surgical repair. So casting

326
00:21:52.160 --> 00:21:56.160
splinting is the accepted approach for uncomplicated
night sticks fractures. But if the fracture

327
00:21:56.200 --> 00:22:02.599
is comminuted over fifty percent displacement,
these types of fractures are suited for better

328
00:22:02.599 --> 00:22:07.440
suited for surgical repair. Okay,
Montezia and Galiazzi fractures. So with Montezia

329
00:22:07.480 --> 00:22:11.279
and Galiazzi fracture is the only thing
I feel you really need to remember that's

330
00:22:11.319 --> 00:22:15.759
normally tested on is what bonus fracture
and dislocated in each. The rest of

331
00:22:15.799 --> 00:22:21.359
the stuff's pretty low yield that you
need to know. So Montezia is going

332
00:22:21.400 --> 00:22:26.119
to be a proximal ulnar fracture accompanied
by a radial head dislocation. So again

333
00:22:26.160 --> 00:22:33.480
Montesia proximal ulnar fracture companied by a
radial head dislocation. Galiazzi is going to

334
00:22:33.480 --> 00:22:38.039
be a radial midshaft fracture associated with
a dislocation of the distal radial ulnar joint.

335
00:22:38.559 --> 00:22:42.160
So both of these are not just
fractures, but they're also dislocations,

336
00:22:42.319 --> 00:22:47.440
and you need both to meet the
criteria for either a Montezia or a Galiazzi

337
00:22:47.480 --> 00:22:49.920
fracture, you need to have both
a fracture and the dislocation. Diagnosis is

338
00:22:49.960 --> 00:22:52.759
going to be done with an X
ray. Treatment is going to be surgical

339
00:22:52.799 --> 00:22:56.640
repair. Generally, in both of
Montezia and a Galiazzi fracture, treatment is

340
00:22:56.680 --> 00:23:02.880
almost always going to be surgical.
Montega fractures in particularly are almost in particular,

341
00:23:02.920 --> 00:23:07.039
almost always unstable and always require surgical
treatment. Most of the time this

342
00:23:07.119 --> 00:23:11.079
is going to be with an RIF
open reduction internal fixation. So again the

343
00:23:11.160 --> 00:23:15.279
highest yield thing is remembering which fracture
and dislocation is of what bone in each

344
00:23:15.279 --> 00:23:19.319
condition. So the way that you
remember that is by remembering gruesome murder.

345
00:23:19.640 --> 00:23:25.960
So gruesome murder helps to remember which
bonus fracture and then secondly which is dislocated,

346
00:23:26.000 --> 00:23:30.839
So remember gruesome murder. So for
Montigia and Galiazzi fracture, so gruesome

347
00:23:30.079 --> 00:23:36.200
The first three letters in gruesome are
gru, so the G stands for Galiazzi,

348
00:23:36.319 --> 00:23:40.119
the R stands for radius fracture,
and then the U stands for owner

349
00:23:40.200 --> 00:23:45.200
dislocation. So that's in gruesome Galiazzi
our radius fracture, and then the U

350
00:23:45.279 --> 00:23:48.000
stands for owlnar dislocation. I know, technically I said this is a dislocation

351
00:23:48.079 --> 00:23:52.200
or instability of the radio owner joint, not specifically in older dislocation, but

352
00:23:52.279 --> 00:23:56.240
in most cases, what we actually
see is this dorsally displaced owner head,

353
00:23:56.440 --> 00:24:00.240
which will be very evident on X
ray, so you'll see the owner hopped

354
00:24:00.240 --> 00:24:03.839
out of place. So remember gruesome
gru Galiazzi, radius fracture, owner dislocation,

355
00:24:04.359 --> 00:24:07.279
and then murder the second part of
the sentence. The first three letters

356
00:24:07.279 --> 00:24:11.720
are Mu, R, and that
stands for M stands for Montezia, U

357
00:24:11.880 --> 00:24:15.119
stands for ulnar fracture, and then
the R stands for radial head dislocation.

358
00:24:15.160 --> 00:24:19.599
So again remember gruesome murder. First
three letters of gruesome, Galiazzi, radius

359
00:24:19.640 --> 00:24:23.599
fracture, owner dislocation, first three
letters of murder. Help you remember Montezia,

360
00:24:23.920 --> 00:24:29.480
ulner fracture and radio head dislocation.
Okay, radial head subluxation This is

361
00:24:29.480 --> 00:24:33.119
also known as a nursemaide elbow.
It's important to know this because not only

362
00:24:33.160 --> 00:24:36.960
may it come up on an exam, but if you wind up working anywhere

363
00:24:36.960 --> 00:24:40.279
with pediatric patients, you're going to
see this very frequently. So what happens

364
00:24:40.279 --> 00:24:44.640
here you have the movement of the
head of the radius that slips under the

365
00:24:44.680 --> 00:24:48.480
annular ligament. So we talked about
this before, but the ligaments and children

366
00:24:48.480 --> 00:24:51.599
are much more lax and weak.
So in a child, when you have

367
00:24:51.680 --> 00:24:55.200
this sudden pulling on the arm,
pulling on the distill radius, the annular

368
00:24:55.240 --> 00:24:57.519
ligament, which is a band of
fibers that encircles the head of the radius,

369
00:24:57.759 --> 00:25:00.799
it slips right over the head the
radius, and then this slides into

370
00:25:00.839 --> 00:25:04.920
the radio humeral joint where it becomes
trapped and these children start to have pain.

371
00:25:06.559 --> 00:25:10.880
It's most common in one to four
years of age, so common between

372
00:25:10.920 --> 00:25:14.559
the ages of one to four years. Eighty percent of cases are actually going

373
00:25:14.599 --> 00:25:17.720
to fall within the one to three
year range. So if it's not a

374
00:25:17.720 --> 00:25:19.400
young child in the vignette, it's
not a nursemaid elbow. This is a

375
00:25:19.480 --> 00:25:22.799
one hundred percent going to be a
young child. The reason we see this

376
00:25:23.000 --> 00:25:27.319
more frequently in young children not adults. Is because the attachment of the annular

377
00:25:27.440 --> 00:25:32.079
ligament is much weaker and children than
adults. As the child grows, the

378
00:25:32.079 --> 00:25:36.079
attachment between the annular ligament and the
radius becomes thicker and stronger, and it

379
00:25:36.079 --> 00:25:40.039
prevents the subluxcation. Usually by the
age of five years, the ligament is

380
00:25:40.039 --> 00:25:42.359
going to be strong enough and thick
enough that it's highly unlikely to tear to

381
00:25:42.400 --> 00:25:45.519
become displaced. So generally you're looking
for a child one to four years.

382
00:25:45.720 --> 00:25:48.680
I'm not saying it's impossible to see
this like in a seven or eight year

383
00:25:48.680 --> 00:25:52.000
old, but probably not very common
at all. Look for a very young

384
00:25:52.079 --> 00:25:56.480
child. In the vignette mechanism of
injury, this is going to be a

385
00:25:56.519 --> 00:26:02.759
polling injury. So the classic mechanism
for a radiohead subluxation is going to be

386
00:26:02.960 --> 00:26:06.000
consisting of a pull injury. So, for instance, a parent grabs the

387
00:26:06.079 --> 00:26:08.920
arm of the child to prevent them
from falling, maybe the child's being swung

388
00:26:08.960 --> 00:26:12.400
by the forearms or during play with
their siblings or parents. You're looking for

389
00:26:12.480 --> 00:26:15.400
some type of pull or tug on
the arm. And even yet, the

390
00:26:15.440 --> 00:26:18.640
reason why this is actually called a
nursemaid elbow was because back in the day,

391
00:26:19.839 --> 00:26:23.200
when nursemaids as they were called back
in the day, took care of

392
00:26:23.279 --> 00:26:26.960
children, they were often blamed for
causing this injury by tugging on the child's

393
00:26:27.000 --> 00:26:30.519
arm. So look for that pulling
or tugging of the arm. Diagnosis is

394
00:26:30.559 --> 00:26:33.440
going to be clinical. So a
majority of the time this is a clinical

395
00:26:33.480 --> 00:26:37.200
diagnosis. They had a pulling or
tugging type injury. They fit the age

396
00:26:37.279 --> 00:26:41.000
range on physical exam, they don't
have any bony tenderness, no deformity,

397
00:26:41.000 --> 00:26:45.640
no swelling. Usually that's all you
need to make the diagnosis. Imaging is

398
00:26:45.720 --> 00:26:51.039
usually not necessary. Now treatment there's
very few things in medicine where we can

399
00:26:51.039 --> 00:26:53.759
fix someone's problems and a matter of
seconds and make them feel completely back to

400
00:26:53.839 --> 00:26:59.079
normal. But this is one of
those few circumstances. So with treatment it's

401
00:26:59.079 --> 00:27:02.079
going to be a close reduction,
and there's a couple of ways to do

402
00:27:02.160 --> 00:27:04.559
this. The technique preferred by up
to date, which has a higher rate

403
00:27:04.559 --> 00:27:10.880
of success, is reduction with hyperpronation
rather than the other alternative, which is

404
00:27:10.920 --> 00:27:17.000
this supernation flection method. So the
way you do the hyperpronation method is going

405
00:27:17.039 --> 00:27:19.400
to be you hold the child's arm
at the elbow. You place pressure with

406
00:27:19.440 --> 00:27:23.319
a finger on the radio head and
then the other hand holds the discal forum

407
00:27:23.359 --> 00:27:26.400
and you hyper pronate the forum,
so you turn the arm all the way

408
00:27:26.440 --> 00:27:30.240
over and in a lot of times
you're going to feel this click or this

409
00:27:30.319 --> 00:27:33.000
pop over the radial head. But
it reduces and then these kids within minutes

410
00:27:33.000 --> 00:27:36.559
are going to be back to normal
and playing and smiling in most cases,

411
00:27:36.599 --> 00:27:41.119
at least from my experience. So
for radial head subluxation, main takeaways here

412
00:27:41.359 --> 00:27:45.200
looking for a child under five looking
for a pulling injury. It's a clinical

413
00:27:45.200 --> 00:27:48.880
diagnosis and treatment is going to be
with a closed reduction, very straightforward.

414
00:27:48.039 --> 00:27:52.799
All right, Medio versus lateral epicond
litis. These are really simple, there's

415
00:27:52.880 --> 00:27:55.839
very little to know. But the
reiblement is you can easily get them mixed

416
00:27:55.920 --> 00:27:59.000
up and miss out on an easy
exam question. So let's go over what

417
00:27:59.039 --> 00:28:02.279
you need to know the little differences
between the two and in the monic to

418
00:28:02.319 --> 00:28:07.000
remember them. So, lateral epicondolitus, which is also known as tennis elbow,

419
00:28:07.680 --> 00:28:11.079
it's a frequent complaint of tennis players, actually up to fifty percent of

420
00:28:11.119 --> 00:28:15.119
tennis players. It's mostly due to
poor technique. So it's an overuse injury

421
00:28:15.160 --> 00:28:21.799
of the origin of the common extensor
tendon which leads to tendinosis and inflammation of

422
00:28:21.839 --> 00:28:27.880
the extensor carpi radialis brevis precipitated by
repetitive wrist extension. And the reason why

423
00:28:27.880 --> 00:28:33.319
you have painted the lateral epicondyle is
because the lateral epicondo of the elbow is

424
00:28:33.359 --> 00:28:37.039
the bony origin for the wrist extensors. So who are you looking out for?

425
00:28:37.160 --> 00:28:40.359
This is going to be of course
tennis players. Like I said before,

426
00:28:40.599 --> 00:28:44.440
tennis is the most common sport to
cause lateral epicondolitus. It can be

427
00:28:44.440 --> 00:28:48.079
seen in other sports squash, badminton, as well as certain occupations like carpenters,

428
00:28:48.079 --> 00:28:51.880
bricklayers, seamstresses. But for the
sake of the exam, be looking

429
00:28:51.880 --> 00:28:56.279
for your tennis player, and on
exam you're going to be looking for localized

430
00:28:56.319 --> 00:29:03.240
tenderness over the lateral epicondyle and pain
with resisted wrist extension. So lateral epicondo

431
00:29:03.319 --> 00:29:10.400
lightis associated with extensive extension and extensor
so pain with risk extension. On physical

432
00:29:10.400 --> 00:29:17.160
exam, overuse of the extensor tendon, the extensor carpi radialist brevis precipitated from

433
00:29:17.160 --> 00:29:21.920
repetitive risk extension. Remember what I
keep saying, extension extensor, lateral epikind

434
00:29:21.920 --> 00:29:26.039
of lightis. Remember extension extension extension
That's how they're going to get you on

435
00:29:26.119 --> 00:29:27.720
an exam question if you don't remember
this and you get it mixed up with

436
00:29:27.839 --> 00:29:33.240
medial epicind of lightis, which is
the opposite involving flexion rather than extension treatment.

437
00:29:33.440 --> 00:29:37.960
It's going to be conservative and SAIDs
activity modification, steroid injections, counter

438
00:29:38.039 --> 00:29:42.359
force braces, not anything really important
to know here. Medial epicondo lightis.

439
00:29:42.440 --> 00:29:48.000
So this is also known as golfer's
elbow. It is an overuse injury involving

440
00:29:48.039 --> 00:29:53.839
the proximal tenders of the pronator,
terrace and flex or carpi. Radialists probably

441
00:29:53.880 --> 00:29:59.559
mispronouncing all of these overt imachin I
am. It's primarily due to repetitive forceful

442
00:30:00.160 --> 00:30:03.880
forearm pronation and risk flection. So
this is the opposite and lateral we were

443
00:30:03.880 --> 00:30:08.519
talking all about extension extens or tendence
down medial It's all about the flection flex

444
00:30:08.599 --> 00:30:14.559
or carpon radialis caused by repetitive risk
flection and just like we talked about before

445
00:30:14.799 --> 00:30:18.079
in lateral epicondolitis, the reason why
you have pain in the medial epicondo in

446
00:30:18.119 --> 00:30:23.519
this case is because the medial epicondyle
is the common origin of the forearm flexor

447
00:30:23.599 --> 00:30:27.759
and and the pronator muscles. So
in the vignette you're probably going to get

448
00:30:27.759 --> 00:30:33.839
a golfer medial epicondolitis, as we
know is called golfer's elbow. But interestingly

449
00:30:34.000 --> 00:30:38.359
enough, around ninety percent of cases
are actually non sports related, in particular

450
00:30:38.759 --> 00:30:45.359
occupational settings with repeated forceful gripping during
heavy labor like plumbers, carpenters, construction

451
00:30:45.359 --> 00:30:48.880
workers. But for the exam,
I probably remember and focus on your golfers

452
00:30:48.559 --> 00:30:53.240
on physical exam. They're going to
have localized tenderness over the medial epicondyle and

453
00:30:53.359 --> 00:30:59.759
pain with resisted wrist flection, so
flection flection, flection treatment is going to

454
00:30:59.799 --> 00:31:03.680
be the same as in lateral conservative
and SAIDs activity modifications, steroid injections,

455
00:31:03.720 --> 00:31:07.359
counterforce braces. Nothing to memorize there, all right, So the main thing

456
00:31:07.640 --> 00:31:14.279
to remember for both later epicondolitis remember
tennis players pain with wrist extension against resistance

457
00:31:14.599 --> 00:31:21.359
problem with the extensor tendence caused by
repetitive risk extension, Lateral should equal extension

458
00:31:21.480 --> 00:31:26.160
in your mind. Now, medial
epicondolitis golfers pain with risk flection against resistance

459
00:31:26.440 --> 00:31:30.759
problem with the flex or muscles caused
by repetitive wrist flection. Medial should equal

460
00:31:32.119 --> 00:31:34.720
flection in your mind. If you
can remember one by method of exclusion,

461
00:31:34.759 --> 00:31:37.799
you can remember the other. And
here's how you're going to do that.

462
00:31:37.039 --> 00:31:41.960
So with medial epicondolitis aka golfer's elbow, I want you to remember mini golf

463
00:31:42.079 --> 00:31:47.759
is fun. The M and mini
helps you remember this is medial epicondel involved

464
00:31:48.000 --> 00:31:52.240
in golfer's elbow golf obviously because this
is also known as golfer's elbow. And

465
00:31:52.319 --> 00:31:56.440
then the F and fun helps you
remember this involves flexion, whether it's pain

466
00:31:56.480 --> 00:32:00.839
with risk, flection against resistance on
exam or the fact that the flex or

467
00:32:00.920 --> 00:32:05.759
carpyrradialis or that it's caused from repetitive
flection. Mini golf is fun. Mini

468
00:32:06.000 --> 00:32:08.240
stands for media up a condal golfer
golfer's elbow, and F and fun for

469
00:32:08.279 --> 00:32:13.599
reflection flexor. And then you know
ladder up condolitis is the opposite problems with

470
00:32:13.720 --> 00:32:16.160
extension not flection. So remember mini
golf is fun on your exam. When

471
00:32:16.160 --> 00:32:20.079
you get a question on one of
these, because you're very you very likely

472
00:32:20.119 --> 00:32:22.480
will. And honestly, I had
so much trouble remembering which was which until

473
00:32:22.519 --> 00:32:25.359
I came up with anemonic, and
now I never forget. So remember mini

474
00:32:25.440 --> 00:32:29.680
golf is fun. Okay. Next, I wanted to talk about two conditions

475
00:32:29.720 --> 00:32:32.119
that are also really easy to get
mixed up on an exam, and they

476
00:32:32.119 --> 00:32:37.720
can have similar presentations, and then
I'll teach you some ways to help differentiate.

477
00:32:37.960 --> 00:32:39.880
So that's going to be cubital tunnel
syndrome and carpal tunnel syndrome. So

478
00:32:39.960 --> 00:32:45.960
let's start with cubital tunnel syndrome,
which is ulnar neuropathy. It's a compressive

479
00:32:45.000 --> 00:32:51.640
neuropathy of the ulnar nerve caused by
compression in the medial elbow. Clinical manifestations,

480
00:32:51.680 --> 00:32:54.279
this is the highest yield thing to
know right here. So paristhesia of

481
00:32:54.319 --> 00:33:00.200
the fifth finger and half of the
fourth so numbness tingling in the older nerve

482
00:33:00.240 --> 00:33:02.960
distribution. That's going to be your
most common initial complaints. So paristesia is

483
00:33:04.119 --> 00:33:07.240
the small finger and older half of
the ring finger. It's also possible to

484
00:33:07.279 --> 00:33:10.920
have radiating pain down from the elbow. Usually you're looking for paristesis and the

485
00:33:10.920 --> 00:33:15.160
little finger and half of the ring
finger. That's the key to remember here.

486
00:33:15.799 --> 00:33:17.640
So you're going to get a question, the patient's gonna have some tingling

487
00:33:17.640 --> 00:33:21.880
in their hand. The answer choices, they're going to have both cubital tunnel

488
00:33:21.880 --> 00:33:25.119
syndrome and carpal tunnel. You need
to remember which has which nerve distribution,

489
00:33:25.160 --> 00:33:29.200
and this is the way that you
remember it. So cubital tunnel starts at

490
00:33:29.200 --> 00:33:31.440
the little finger, the pinky and
half of the ring finger. Carpal tunnel

491
00:33:31.519 --> 00:33:35.400
starts at the big finger, the
opposite end the thumb or the first digit,

492
00:33:35.759 --> 00:33:37.200
all the way through the other half
of the ring finger. So how

493
00:33:37.240 --> 00:33:40.640
do you remember that for the exam, Well, cubital starts with the letters

494
00:33:40.799 --> 00:33:46.720
sub sub cub and as we know, cubs are little bears, bear cubs.

495
00:33:46.839 --> 00:33:51.440
And then help to remember that cubital
cub is the one that starts at

496
00:33:51.480 --> 00:33:53.759
the little finger, the pinky,
and by method of exclusion, you crewmember,

497
00:33:53.799 --> 00:33:58.079
carpal tunnel started at the other end, the big finger, the thumb.

498
00:33:58.240 --> 00:34:00.079
So as soon as you see cubital
tunnels syndrome. Think of a cub,

499
00:34:00.200 --> 00:34:04.160
cub a little bear. Remember this
is the one that starts at the

500
00:34:04.200 --> 00:34:07.640
little finger. Carpal tunnel starts at
the other hand, at the big finger,

501
00:34:07.720 --> 00:34:09.320
the thumb. So I remember for
the exam, and I got the

502
00:34:09.360 --> 00:34:14.079
question right because I remember cubs or
little animals. Cubical tunnel syndrome starts at

503
00:34:14.079 --> 00:34:17.800
the little finger. So on physical
exam, there's a few different provocation tests

504
00:34:17.800 --> 00:34:22.400
for cubital tunnel, but the one
physical test I'd remember for this is the

505
00:34:22.440 --> 00:34:24.280
Tannell sign, as this is the
one you'll hear most often, or the

506
00:34:24.320 --> 00:34:30.119
Tennel test. So this is just
percussion or tapping over the ular nerve and

507
00:34:30.159 --> 00:34:34.199
the umlar groove of the elbow at
the cubital tunnel. So when you do

508
00:34:34.239 --> 00:34:37.119
this, if they have parastesia and
the fourth and the fifth finger, that's

509
00:34:37.119 --> 00:34:40.480
a positive Tennell sign. There's also
a Tannel sign or test and carpal tunnel

510
00:34:40.519 --> 00:34:43.760
as well, so don't get them
mixed up. We'll go over that.

511
00:34:43.880 --> 00:34:47.719
Next treatment for cubital tunnel is going
to be conservative for a surgical Initially,

512
00:34:47.840 --> 00:34:53.400
start with activity modification. If this
is from some sort of repetitive trauma or

513
00:34:53.400 --> 00:34:58.960
occupational cause. You can use splints
and SAIDs. Surgery is really only going

514
00:34:58.960 --> 00:35:02.199
to be for severe or progressive symptoms. Now let's talk about carpal tunnel syndrome.

515
00:35:02.280 --> 00:35:06.679
So this is compression of the media
nerve as it travels through the carpal

516
00:35:06.679 --> 00:35:10.400
tunnel. So cubital tunnel syndrome was
ulder nerve compression. Carpal tunnel is media

517
00:35:10.480 --> 00:35:15.880
nerve compression. Different nerves affected,
different nerve distribution, and different area affected.

518
00:35:15.719 --> 00:35:19.880
So again this is the really important
thing you need to remember to differentiate

519
00:35:19.880 --> 00:35:22.519
the two. So in carpal tunnel
syndrome, you're going to have pain and

520
00:35:22.599 --> 00:35:25.440
parasthesia of the first three digits and
half of the fourth. So a lot

521
00:35:25.480 --> 00:35:28.840
of times the pain is going to
be present at night. It's going to

522
00:35:28.920 --> 00:35:32.559
wake them from sleep, the hallmark
presentation paint or parasthesia, and the distribution

523
00:35:32.840 --> 00:35:37.119
that includes the media nerve territory,
so thumb, the first digit all the

524
00:35:37.159 --> 00:35:40.719
way through the fourth digit, the
radial or lateral half, so first,

525
00:35:40.719 --> 00:35:45.079
second, and third and half of
the fourth fingers different than cubital tunnel,

526
00:35:45.079 --> 00:35:46.760
which we know started at the opposite
end of the hand at the little finger,

527
00:35:47.039 --> 00:35:50.840
fifth digit and half of the other
half of the fourth digit on the

528
00:35:50.880 --> 00:35:53.280
ulner side, so carpal tunnel fingers
one, two, three and a half

529
00:35:53.280 --> 00:35:58.280
of four, cubital tunnel finger five
and half of four. Other things to

530
00:35:58.360 --> 00:36:00.800
look out for, although not as
important. They may have weakness or clumsiness

531
00:36:00.800 --> 00:36:05.480
when using their hands. They may
have difficulty holding objects, turning keys or

532
00:36:05.519 --> 00:36:08.599
door knobs, buttoning clothing. They
may have atrophy of the theen or eminence

533
00:36:08.599 --> 00:36:12.199
in advanced cases. So those are
just some other things to be aware of

534
00:36:12.639 --> 00:36:16.360
now. Risk factors. There's a
lot of risk factors for carpal tunnel obviously

535
00:36:17.000 --> 00:36:22.039
repetitive hand and risk use, particularly
with some occupations authritis, obesity, female

536
00:36:22.079 --> 00:36:27.360
gender, but the ones that I
would focus on for the exam are pregnancy.

537
00:36:28.000 --> 00:36:30.119
Third trimester is usually where the symptoms
will start to manifest. It can

538
00:36:30.159 --> 00:36:35.360
be diagnosed earlier on than this though
diabetes molitis. Both type one and type

539
00:36:35.360 --> 00:36:39.800
two diabetes can be associated with carpal
tunnel syndrome and then hypothyroidism. So hypothyroidism

540
00:36:39.880 --> 00:36:45.679
contributes to the development of carpal tunnel
by increasing peripheral tissue aedema. So those

541
00:36:45.679 --> 00:36:47.840
are usually the ones you're going to
get tested on. Let's talk about one

542
00:36:47.920 --> 00:36:52.360
other high yield area for carpal tunnel
that you'll definitely need to know for your

543
00:36:52.360 --> 00:36:57.280
OSCIS. That's your physical exam tests. So there's a few revocative maneuvers you'll

544
00:36:57.320 --> 00:36:59.840
hear about for carpal tunnel. The
two that you need to know, the

545
00:36:59.840 --> 00:37:02.519
one that frequently come up are the
Tannelle test and the Phalin test. So

546
00:37:02.599 --> 00:37:06.800
Tannell test just like in cubital tunnel, or we tapped on the cubital tunnel,

547
00:37:07.159 --> 00:37:09.840
now we're tapping or percussing over the
carpal tunnel. It's easy to get

548
00:37:09.840 --> 00:37:13.639
these physical exam tests mix up.
So I used to remember the t and

549
00:37:13.639 --> 00:37:16.519
Tannell stood for tapping because it helps
you remember the test is performed. So

550
00:37:16.559 --> 00:37:21.519
you're tapping, tapping, tapping on
the median nerve over the carpal tunnel.

551
00:37:21.639 --> 00:37:25.480
Positive test is defined as pain or
parasthesia of the median innervated fingers, remember

552
00:37:25.559 --> 00:37:29.320
first finger through half of the fourth
and then we have the Phalin test.

553
00:37:29.400 --> 00:37:32.239
So the Phalin test is performed by
having the patients bring the dorsal surfaces so

554
00:37:32.360 --> 00:37:37.360
the back of the hands against each
other, pushing them up against each other

555
00:37:37.360 --> 00:37:40.440
at the top of the hands push
them together and this provides hyperflection of the

556
00:37:40.480 --> 00:37:44.480
risks while the elbows are going to
remain flexed. You do this for one

557
00:37:44.519 --> 00:37:47.639
minute straight and a positive test is
going to be pain or parastesia again and

558
00:37:47.719 --> 00:37:52.519
the media innervated fingers. The way
that I used to remember this is if

559
00:37:52.519 --> 00:37:54.000
you actually look at a picture of
the phalin test. When you flop your

560
00:37:54.000 --> 00:37:58.039
hands down like this, it looks
like your hands have just kind of like

561
00:37:58.159 --> 00:38:00.639
flopped over. They've fallen over.
So when I see phalin tests, I

562
00:38:00.679 --> 00:38:06.119
just remember that your hands have phalin
over. Because the hands are flopped over,

563
00:38:06.159 --> 00:38:09.199
they look like they've fallen or phalin
over. So I just remember phalin.

564
00:38:09.320 --> 00:38:15.440
Your hands have phalin or fallen over. Tannell. Remember t for tannel

565
00:38:15.519 --> 00:38:20.159
and tapping tapping on the media nerve
phalin. Hands have phalin over pressed up

566
00:38:20.159 --> 00:38:22.480
against each other for a minute.
So those are the two provocation tests i'd

567
00:38:22.480 --> 00:38:27.400
know for a carpal tunnel. Also
be aware there are nerve conduction studies,

568
00:38:27.400 --> 00:38:30.840
electromiography. Those are some other diagnostic
tests can help you with the diagnosis,

569
00:38:30.079 --> 00:38:34.079
but focus on those physical exam tests. I talk about the tannel and the

570
00:38:34.079 --> 00:38:38.880
phalin treatment. Conservative or surgical so
splinting glucocorticoids, whether it's PO or VA

571
00:38:38.880 --> 00:38:43.039
injections are going to be useful for
symptom relief, and up to two thirds

572
00:38:43.039 --> 00:38:46.159
of patients with mild or monitor carpal
tunnel severe refractory cases, of course,

573
00:38:46.199 --> 00:38:51.920
surgical decompression may be needed. Let's
move on to our scafloid or our navicular

574
00:38:51.920 --> 00:38:57.199
fracture. This is your most common
carpal fracture. So scaffoid fractures are the

575
00:38:57.199 --> 00:39:00.440
most common carpon bone fracture. They're
seen in around sixty to seventy percent of

576
00:39:00.559 --> 00:39:05.039
all carbal fractures, so that's an
important one to know. That's often tested

577
00:39:05.119 --> 00:39:08.880
on mechanism. Another foocher so foosh
fallen an outstretched hand will likely be the

578
00:39:08.920 --> 00:39:14.719
cause of a scafoid fracture, or
really any other injury that involves an axial

579
00:39:14.760 --> 00:39:16.880
load placed on the wrist. On
physical exam, they're obviously gonna have some

580
00:39:16.880 --> 00:39:20.559
pain in the wrist, But where
is the pain going to be. It

581
00:39:20.639 --> 00:39:22.639
gonna be a few different places.
It can be distal to the listers,

582
00:39:22.639 --> 00:39:27.559
tubercle, the volar prominence. But
the only one that you have to know,

583
00:39:27.679 --> 00:39:30.159
the one that will ninety nine percent
be how it's described in the vignette,

584
00:39:30.400 --> 00:39:36.039
will be pain or tenderness at the
anatomic snuffbox. Know this for the

585
00:39:36.079 --> 00:39:40.000
exam. As soon as you hear
anatomic snuffbox tenderness, be thinking scaffoid fracture

586
00:39:40.199 --> 00:39:45.599
always, so any tenderness in the
snuffbox should be treated as a scaffoid fracture

587
00:39:45.960 --> 00:39:50.440
until proven otherwise. The snuffbox is
located just proximal to the base of the

588
00:39:50.519 --> 00:39:53.239
thumb. Now diagnosis, you're gonna
order a risk X ray. Obviously you

589
00:39:53.320 --> 00:39:58.400
want to include a scaffoid view.
The thing about scaffoid fractures, though,

590
00:39:58.440 --> 00:40:00.760
is that they can be missed on
X ray. Is pretty often X rays

591
00:40:00.800 --> 00:40:05.360
taken soon after the injury can be
missed up to fifty four percent of cases,

592
00:40:05.440 --> 00:40:08.679
so half the time. So if
the history and physical exam findings are

593
00:40:08.719 --> 00:40:14.239
suspicious for a scaffoid fracture but the
X rays are negative, you're often gonna

594
00:40:14.280 --> 00:40:17.320
have to send these patients for a
CTRMRI because if you miss a scaffoid fracture,

595
00:40:17.679 --> 00:40:21.480
the scaffoid, unfortunately has a crappy
blood supply. And can be at

596
00:40:21.519 --> 00:40:24.400
risk for non union osteon necrosis.
So if it fits the picture of a

597
00:40:24.400 --> 00:40:28.719
scaffoid fracture but the X rays are
negative, you still treat it like a

598
00:40:28.760 --> 00:40:34.199
fracture until you can definitively say otherwise. Treatment thumb spike up. Treatment for

599
00:40:34.440 --> 00:40:37.760
majority of patients with a non displaced
fracture or those patients who have negative X

600
00:40:37.840 --> 00:40:43.400
rays but have snuffbox tenderness, you
give these patients a thumb spike until you

601
00:40:43.400 --> 00:40:45.960
can roll out a fracture with the
scaffoid. Again, treat it like a

602
00:40:46.000 --> 00:40:51.880
fracture until you know it ain't surgery. If it's a displaced fracture or there's

603
00:40:51.920 --> 00:40:55.400
neurovascular compromise, surgical repair will be
appropriate, but in most cases it's going

604
00:40:55.480 --> 00:40:59.119
to be your thumb spike of splint
will be the answer. Choice. All

605
00:40:59.199 --> 00:41:02.599
right, let's talk about distal radius
fractures are Colleagues versus our Smith fractures.

606
00:41:02.719 --> 00:41:06.159
There's really just a few things to
know here, mainly just being able to

607
00:41:06.159 --> 00:41:09.800
differentiate between a Collie and a Smith
fracture mechanism. Again, Foush, most

608
00:41:09.840 --> 00:41:14.840
common mechanism of the distal radius fracture
is going to be falling on an outstretched

609
00:41:14.840 --> 00:41:19.920
hand. More of the more often
the risk is going to be extended in

610
00:41:19.960 --> 00:41:23.119
a Collie fracture injury, and often
you'll see it's flexed in a Smith fracture.

611
00:41:23.199 --> 00:41:29.039
So just a little side note there
on physical exam, collies fractures have

612
00:41:29.159 --> 00:41:32.559
what's known as a dinner fork deformity, and this is due to the dorsal

613
00:41:32.639 --> 00:41:37.880
displacement of the distal fragment. Collige
type fractures are often said to have this

614
00:41:37.960 --> 00:41:39.360
dinner fork appearance. If you want
to see some pictures that have it in

615
00:41:39.360 --> 00:41:44.559
the YouTube video. And then Smith
fractures are sometimes described as a garden spade

616
00:41:44.599 --> 00:41:47.440
deformity. Don't go too crazy memorizing
these names dinner fork, garden spade,

617
00:41:47.480 --> 00:41:51.360
because they're likely not going to use
these buzzwords. You just need to remember

618
00:41:51.440 --> 00:41:53.239
that one has dorsals displacement, the
other has eventual displacement. I'm going to

619
00:41:53.280 --> 00:41:57.719
give you anemonic for that in a
second, but remember, I guess Smith

620
00:41:57.760 --> 00:42:00.960
fracture sometimes known as a garden spade
deform me, and that's if you see

621
00:42:00.000 --> 00:42:02.880
like a spade or a shovel and
has this little bump on it, and

622
00:42:02.880 --> 00:42:07.000
that's because of the eventual displacement.
It looks like that anyways. Diagnosis,

623
00:42:07.039 --> 00:42:09.440
this is what you really need to
know. So when you take an X

624
00:42:09.559 --> 00:42:15.079
ray on a Collie's fracture, you're
going to see dorsal displacement of the distal

625
00:42:15.159 --> 00:42:21.320
radius fracture, dorsal displacement of the
disarradius fracture, and a Collie's fracture and

626
00:42:21.440 --> 00:42:25.039
a Smith fracture, you're going to
see volar or palm our displacement of the

627
00:42:25.079 --> 00:42:31.400
distal radius fracture. So remember Collie's
dorsal displacement Smith volar or palm our displacement

628
00:42:31.400 --> 00:42:35.719
of the distal radius fracture. That's
the highest heal thing to know because this

629
00:42:35.880 --> 00:42:38.320
was differentiates the two and it may
be the only difference in the vignette.

630
00:42:38.360 --> 00:42:43.360
So remember Collie's fractured, dorsal angulation
of the radius, Smith fracture of volar

631
00:42:43.360 --> 00:42:46.639
angulation of the radius. The way
that you remember that a Collie's fracture is

632
00:42:46.639 --> 00:42:52.079
dorsal displacement is because a Collie is
actually a breed of a dog. Nice

633
00:42:52.119 --> 00:42:55.199
and conveniently it's the same dog that
Lassie was on that show a number of

634
00:42:55.280 --> 00:42:59.679
years ago. So a Collie dog
when you think of a Collie fracture,

635
00:42:59.719 --> 00:43:02.320
Think of a Collie dog and the
first two letters in dog are the first

636
00:43:02.320 --> 00:43:08.079
two letters in dorsaldo. So this
helps you remember Collie's fractures are doo dorsally

637
00:43:08.559 --> 00:43:14.920
dorsally alculated radial fractures and by method
of exclusion, smith is the opposite volar

638
00:43:14.960 --> 00:43:19.360
angulated radial fractures. So when you
see collie fracture, think of a Collie

639
00:43:19.400 --> 00:43:24.440
dog doo dorsal dog treatment splint splint, reduction for surgery. So some fractures

640
00:43:24.480 --> 00:43:30.199
can be treated with closed reduction sugar
tongue splint. Other fractures are gonna require

641
00:43:30.199 --> 00:43:35.000
surgical or repair defend, depending on
the severity. Remember your Collie dog dorsal

642
00:43:35.039 --> 00:43:37.719
angulation for your Collie fracture. Just
knowing that one thing may get you the

643
00:43:37.800 --> 00:43:40.960
question. Very likely it will if
you ever forget which side is the dorsal

644
00:43:42.000 --> 00:43:44.920
side of the hand or the risk. Remember the dorsal fin of a dolphin

645
00:43:45.039 --> 00:43:47.360
is on its back. Therefore,
a fracture with dorsal angulation will be angulated

646
00:43:47.360 --> 00:43:51.960
for the back or the top of
the wrist area. All right, lunate

647
00:43:52.039 --> 00:43:55.679
fractures. So, lunate fractures are
pretty uncommon. Their only account for about

648
00:43:55.679 --> 00:43:59.800
four percent of all carpal bone injuries, but they do have some pretty serious

649
00:44:00.000 --> 00:44:04.480
applications if they're not treated. So
mechanism of injury is going to be another

650
00:44:04.559 --> 00:44:08.079
fall on an extended wrist or any
other type of risk hyper extension injuries.

651
00:44:08.280 --> 00:44:12.719
These are going to be your most
common mechanisms of injuries for a lunar fracture

652
00:44:13.119 --> 00:44:15.199
diagnosis, so X ray. Main
thing to know about X rays of the

653
00:44:15.239 --> 00:44:20.800
lunate is that they're missed quite often
with playing radiographs. If there's any clinical

654
00:44:20.800 --> 00:44:24.360
suspicion, it's important to obtain vanced
imaging like ctrmri CT is going to be

655
00:44:24.840 --> 00:44:29.079
preferred. One of the thing that
I wanted to mention because it may come

656
00:44:29.119 --> 00:44:34.079
up if you have a dislocation of
the lunate bone. The lunate gets displaced

657
00:44:34.119 --> 00:44:37.920
and angled in this volar direction and
in comparison to the surrounding structure is the

658
00:44:37.960 --> 00:44:42.960
distal radius metacarpals which are all in
normal alignment. It looks like a tea

659
00:44:43.000 --> 00:44:45.800
cup spilling over on lateral X rays, at least that's what they say,

660
00:44:45.840 --> 00:44:47.880
and it's called a spilled tea cup
sign. So just in case, you

661
00:44:47.880 --> 00:44:52.079
hear that being mentioned at any point
you'll know. This is what can be

662
00:44:52.119 --> 00:44:57.079
seen in a lunate dislocation. So
you need to know a complication a vascular

663
00:44:57.159 --> 00:45:02.320
necrosis and kind box disease. So
aggressive collapse of the lunate mechanism of injury

664
00:45:02.519 --> 00:45:06.920
is really unclear. This is kind
box disease, so it's a progressive collapse

665
00:45:06.920 --> 00:45:12.280
of the lunate mechanism of mechanism is
really unclear. It evolves to appear some

666
00:45:12.519 --> 00:45:15.760
disruption of the blood supply. It's
likely related to undiagnosed fractures of the lunate.

667
00:45:16.480 --> 00:45:22.039
Some other complications with lunar fractures are
complex regional pain, syndrome, osteoarthritis.

668
00:45:22.559 --> 00:45:27.800
Nothing specific I would know for the
treatment for lunar fractures, except in

669
00:45:27.840 --> 00:45:30.960
the case of a dislocation, in
which immediate reduction is really important to prevent

670
00:45:31.000 --> 00:45:36.760
a number of complications. So again
that's your lunate fractures and a little bit

671
00:45:36.800 --> 00:45:39.920
about dislocations. Not a lot to
know there. Let's talk about decoreving tendinopathy

672
00:45:40.039 --> 00:45:45.920
next. So this is a thickening
of the abductor policis longest the apl and

673
00:45:46.079 --> 00:45:52.559
extensor policis brievious, the EPV tendons
and the tunnel or the sheath and the

674
00:45:52.559 --> 00:45:59.679
first extensor compartment. So the abductor
policis longest apl the extensor policies brievious EPB

675
00:45:59.800 --> 00:46:04.880
tends. They pass through this tunnel
called the fibro osseous tunnel from the forearm

676
00:46:05.000 --> 00:46:08.519
into the hand, and any thickening
of these tendons passing through here, or

677
00:46:08.599 --> 00:46:13.679
thickening of the tunnel itself can restrain
the gliding motion through the sheath, which

678
00:46:13.679 --> 00:46:16.159
can lead to the clinical manifestations will
go over. So think of a rope

679
00:46:16.199 --> 00:46:20.480
going through a little hole and the
rope's getting bigger or the holes getting smaller.

680
00:46:20.480 --> 00:46:25.400
Eventually it's going to get stuck.
I don't recommend memorizing abductor polysis longest

681
00:46:25.679 --> 00:46:30.440
an extensor polysis brevist. Those are
hard enough to say. It's just too

682
00:46:30.519 --> 00:46:35.199
much brainpower to memorize those complicated names. What I would recommend memorizing, though,

683
00:46:35.639 --> 00:46:38.239
is apples with extra peanut butter are
delicious, because if you can remember

684
00:46:38.559 --> 00:46:45.880
that apples spelled apl apl as an
APL tendon with extra peanut butter EPB as

685
00:46:45.880 --> 00:46:51.119
an EPB tendon are delicious as in
decore vein tendinopathy. If you can remember

686
00:46:51.159 --> 00:46:55.360
that, that helps you remember your
APL tendon and your EPB tendon are going

687
00:46:55.400 --> 00:47:01.360
to be involved in this decorvein sendingpathy, and this should be enough for you

688
00:47:01.400 --> 00:47:05.400
to be able to pick them out
on the multiple choice question by remembering the

689
00:47:05.480 --> 00:47:07.840
letters involved in the full name.
So if you can remember abductor policies as

690
00:47:07.840 --> 00:47:12.039
long as an extensor policies breathes,
in addition of the twenty thousand other things

691
00:47:12.079 --> 00:47:14.760
you need to for PA school,
more power to you. But I just

692
00:47:14.800 --> 00:47:16.679
remember apples with extra peanut butter are
delicious. That was enough for me to

693
00:47:16.679 --> 00:47:21.840
help pick out the right answer on
my clinical medicine exam. So apples again,

694
00:47:21.920 --> 00:47:25.880
APL, extra peanut butter EPB are
delicious. Decorva intentanopathy. You're done,

695
00:47:27.119 --> 00:47:29.880
all right, So what about the
patient demographic you're looking for? So

696
00:47:30.719 --> 00:47:35.880
you are going to be looking for
women between the ages of thirty to fifty

697
00:47:35.960 --> 00:47:38.320
years of age, and then it's
also prevalent in a subset of women in

698
00:47:38.320 --> 00:47:43.320
the postpartum period. Symptoms usually going
to present about four to six weeks after

699
00:47:43.400 --> 00:47:45.599
delivery. There's a lot of theories
why this happens in the postpartum period,

700
00:47:46.599 --> 00:47:51.480
repetitive motion of the hands required to
lift and hold newborns, hormonal causes for

701
00:47:51.559 --> 00:47:53.880
fluid retention, whatever the case.
In the vignette, you're looking for both

702
00:47:53.920 --> 00:47:59.960
women in the thirty to fifty year
age range and also postpartum women clinical menifs

703
00:48:00.039 --> 00:48:01.679
stations, they're going to have pain
at the radial side of the wrist.

704
00:48:02.000 --> 00:48:06.000
It's going to be worse with thumb
and wrist movement. So the tendons we

705
00:48:06.000 --> 00:48:08.199
went over before, the EPB in
the apple, the EPP in the apple,

706
00:48:08.480 --> 00:48:13.960
the EPB and the APL are responsible
for movement of the thumb, So

707
00:48:14.000 --> 00:48:15.440
you can imagine a lot of the
complaints are going to be related to thumb

708
00:48:15.440 --> 00:48:19.199
movements, and so the EPB and
the APL are responsible for movement of the

709
00:48:19.239 --> 00:48:25.880
thumb. Diagnosis so decavent tendonopathy is
based upon the history of an atraumatic radial

710
00:48:27.000 --> 00:48:30.079
wrist pain and positive physical exam findings, So imaging like X ray is not

711
00:48:30.119 --> 00:48:35.079
really necessary except to rule out other
differentials which may have a similar presentation like

712
00:48:35.119 --> 00:48:39.519
osteoarthritis. The main thing to know
for diagnosis is a physical exam maneuver called

713
00:48:39.559 --> 00:48:45.440
the Finkel steam test. So the
Finkel steam provocation test involves the patient wrapping

714
00:48:45.440 --> 00:48:50.800
their fingers around their thumb, clasping
it in their palm, and then you

715
00:48:50.840 --> 00:48:54.199
apply ulnar deviation to the wrist.
If they have pain over the radio styloid

716
00:48:54.239 --> 00:48:58.519
area so the basic of the thumb, this is considered a positive test.

717
00:48:58.559 --> 00:49:01.800
Again, that's the Finkel Steing test. Treatment thumb spike a splints going to

718
00:49:01.840 --> 00:49:07.119
be the big one and SAIDs google
cordecoid injections. So decoving tendonopathy is generally

719
00:49:07.159 --> 00:49:13.400
non progressive. It's typically self limited, so most of your conservative measures are

720
00:49:13.400 --> 00:49:15.440
going to be your mainstay. Again, thumb spike a splint is commonly used,

721
00:49:15.639 --> 00:49:21.239
and said steroid injection surgery really is
going only to be for refractory cases.

722
00:49:22.519 --> 00:49:25.440
Talk about mallet fingernecks. So this
is a finger deformity caused by traumatic

723
00:49:25.480 --> 00:49:30.920
disruption of the terminal slip of the
extensor tendon at the distal interfalangeal joint,

724
00:49:30.960 --> 00:49:35.119
so the dip joint. So you
have some sort of trauma to the finger,

725
00:49:35.280 --> 00:49:37.400
usually caused by a direct blow to
the tip of the finger, so

726
00:49:37.519 --> 00:49:42.960
maybe a ball striking the fingertip or
the fingertip strikes a hard surface. The

727
00:49:43.039 --> 00:49:47.119
trauma causes a tear in the extensor
tendon at the dip joint. And this

728
00:49:47.199 --> 00:49:52.119
is the tendon that allows you to
extend your fingers so to hold them out

729
00:49:52.159 --> 00:49:53.840
straight, and with this being torn, you can no longer do that,

730
00:49:53.840 --> 00:49:59.000
so the finger remains in this constant
slit state of flexion. Now on physical

731
00:49:59.039 --> 00:50:01.480
exam, they're going to have an
inability to extend the dip joint. This

732
00:50:01.519 --> 00:50:06.000
is going to result in that flex
DP So it's really simple. Patient's going

733
00:50:06.039 --> 00:50:08.039
to have this constant flection of the
dip joint. They're not going to be

734
00:50:08.079 --> 00:50:13.920
able to extend the finger at that
joint. This specific degree of the dip

735
00:50:14.000 --> 00:50:17.760
angulation is often going to reflect the
severity of the tendon disruption. Now diagnosis

736
00:50:19.000 --> 00:50:22.480
X ray. So in some cases
with the mallet finger, you're going to

737
00:50:22.559 --> 00:50:25.480
visualize a bony evulsion of the distal
failings at the site where the tendon attaches.

738
00:50:25.719 --> 00:50:30.239
It just got ripped off during the
injury. It's also possible just to

739
00:50:30.320 --> 00:50:34.480
have just a ligamentous injury with normal
bony anatomy, so you can see either

740
00:50:34.519 --> 00:50:38.679
one. Now, treatment really important
extension splinting of the dip joint for six

741
00:50:38.760 --> 00:50:44.639
to eight weeks with twenty four hours
of maintaining this extension, So the dip

742
00:50:44.760 --> 00:50:50.119
joint must be maintained at full extension
throughout the entire period, including during sleep.

743
00:50:50.440 --> 00:50:52.840
If the joint extension is lost at
any point during this initial treatment period,

744
00:50:52.960 --> 00:50:57.320
the treatment clock is reset and an
additional six weeks of splinting has to

745
00:50:57.360 --> 00:51:00.719
be performed. So if you think
about it, you're keeping those horn tendon

746
00:51:00.840 --> 00:51:04.800
edges aligned. So as soon as
you bend that finger just rips it tears

747
00:51:04.840 --> 00:51:07.639
all over again. So the only
wayfer it to completely heal is to have

748
00:51:07.679 --> 00:51:10.880
that finger in full extension with the
torn ligament aligned for six to eight weeks.

749
00:51:13.000 --> 00:51:16.079
The majority of malletfingers are amendable to
treatment with just splinting. Surgery is

750
00:51:16.079 --> 00:51:21.000
really only going to be a reserved
for large displaced fractures or other complex injuries

751
00:51:21.559 --> 00:51:24.079
that may warrant with surgical referral.
But really focus on splinting as that's going

752
00:51:24.119 --> 00:51:28.280
to be your most common, your
main state treatment. So maletfinger again,

753
00:51:28.360 --> 00:51:32.480
extensor extensor tendon injury. Dip joint
now flex all of the time. They

754
00:51:32.519 --> 00:51:36.960
can't extend it, straighten it out
with a splint done. That's your mallet

755
00:51:36.960 --> 00:51:43.400
finger. Let's talk about ulnar collateral
ligament injury aka a gamekeeper or skiers thumb.

756
00:51:43.440 --> 00:51:46.400
It's an injury caused by damage to
the ulnar collateral ligament of the thumb.

757
00:51:46.719 --> 00:51:52.239
Mechanism of injury is going to be
a forced abduction, abduction and hyper

758
00:51:52.280 --> 00:51:55.159
extension of the thumb. This is
going to be at the metacarpul philangial joint.

759
00:51:55.519 --> 00:51:59.440
That's going to be your most common
cause. So the thumb is stretched

760
00:51:59.719 --> 00:52:04.400
and forced into extreme abduction, whether
this is from a fall and athletic injury

761
00:52:04.760 --> 00:52:08.599
skiing accidents where the thumb strikes a
fixed ski pull, and that's where the

762
00:52:08.719 --> 00:52:14.639
names skier stumb came from or the
name gamekeeper stumb, so not so relevant

763
00:52:14.679 --> 00:52:19.679
anymore. But basically this was this
chronic degeneration of the owlner collateral ligament from

764
00:52:19.719 --> 00:52:22.519
twisting the necks of too many birds
and rabbits. So I'm making that up.

765
00:52:22.519 --> 00:52:25.760
That's what gamekeeper stumb came from.
So yeah, probably won't be the

766
00:52:25.800 --> 00:52:29.800
reason why your patient comes in with
this, but seventy plus years ago when

767
00:52:29.800 --> 00:52:31.960
the name was created, it was
today most often this is going to be

768
00:52:32.000 --> 00:52:37.280
a skiing related injury or another athletic
related injury. On physical exam, you

769
00:52:37.280 --> 00:52:40.920
can pretty much confirm a UCL injury
with your physical exam findings combined with your

770
00:52:40.960 --> 00:52:46.480
appropriate clinical manifestations. So Valgus stress
testing is going to reveal a loss of

771
00:52:46.519 --> 00:52:52.480
integrity of the UCL, so the
injured thumb will have increased laxity of the

772
00:52:52.559 --> 00:52:54.800
MCP joints, so the thumb you're
going to be able to pull much further

773
00:52:54.880 --> 00:53:00.360
away compared with the uninjured thumb.
When Valgus stress is applied, the test

774
00:53:00.400 --> 00:53:05.039
is positive and the patient has the
classic clinical manifestations paintings ascerbated by thumb extension

775
00:53:05.440 --> 00:53:08.360
or abduction, and swelling along the
owner aspect of the thumb at the MCP

776
00:53:08.480 --> 00:53:13.360
joint. This is going to help
to confirm the diagnosis, but you also

777
00:53:13.400 --> 00:53:16.239
want to get an X ray of
the thumb to rule out any possible bony

778
00:53:16.280 --> 00:53:22.039
evulsion, fractures and definitive diagnosis can
be made with an MRI or ultrasound,

779
00:53:22.039 --> 00:53:25.320
but generally it's not necessary treatment.
Thumb spike is splint so a mobilization with

780
00:53:25.360 --> 00:53:30.199
the thumb spika will be sufficient in
most patients. Some patients with a complete

781
00:53:30.199 --> 00:53:36.920
tear or patients that don't respond to
conservative therapy may require surgical intervention. Boxer's

782
00:53:36.960 --> 00:53:39.800
fracture. This one's pretty much straightforward. It's a fracture of the fifth metacarpal

783
00:53:39.880 --> 00:53:45.559
neck. Generally the fifth metacarpal neck
fractures when we're talking about boxer fractures.

784
00:53:45.760 --> 00:53:49.639
Occasionally sometimes we'll hear being referred to
fractures of the fourth metacarpal as well.

785
00:53:49.679 --> 00:53:53.880
But now it's common mechanism of injury
direct trauma to a clenched fist. So

786
00:53:53.960 --> 00:53:58.760
obviously the most common situation where you're
gonna have direct trauma to a closed finch

787
00:53:59.039 --> 00:54:04.079
or just say a clenched fist would
be punching something, so that's going to

788
00:54:04.119 --> 00:54:07.320
be the most common cause. So
whether they're punching a wall, a solid

789
00:54:07.360 --> 00:54:10.320
object, a face, that's why
it's called a boxer's fracture. In reality,

790
00:54:10.360 --> 00:54:15.800
though experienced boxers actually rarely sustained this
type of fracture. It's more the

791
00:54:15.840 --> 00:54:20.400
wild roundhouse punching motion that's common in
street fights that cause this, Or someone

792
00:54:20.400 --> 00:54:22.920
who punched a brick wall who's going
to come in with this type of fracture,

793
00:54:22.239 --> 00:54:25.280
rather than in a floyd Mayweather who's
trained to punch the right way.

794
00:54:27.440 --> 00:54:30.639
Diagnosis are going to be made with
X ray so plaine radiographs of the hand.

795
00:54:30.880 --> 00:54:35.000
This is going to establish the diagnosis
of a metacarpal neck fracture and this

796
00:54:35.039 --> 00:54:38.239
will also help to determine the degree
of the fracture angulation. So treatment is

797
00:54:38.280 --> 00:54:44.519
going to be with a mobilization with
an gut or splint. So gut or

798
00:54:44.519 --> 00:54:49.320
splint is going to be It's going
to be used to mobilize fractures of both

799
00:54:49.360 --> 00:54:52.800
the fourth and the fifth metacarpal necks. If the patient does have an open

800
00:54:52.880 --> 00:54:58.880
fracture, a severely common unit fracture, or if they have significant angulation normally

801
00:54:58.920 --> 00:55:01.360
over thirty to four where your degrees, this is going to require surgical consult

802
00:55:01.840 --> 00:55:06.000
Otherwise, immobilize it with a splint. All right, let's finish up with

803
00:55:06.039 --> 00:55:08.079
something a little bit different, and
because I feel like I can no longer

804
00:55:08.119 --> 00:55:12.760
talk, keep getting stumbling on my
words, let's finish up with complex regional

805
00:55:12.760 --> 00:55:16.199
pain syndrome. So this is an
array of painful conditions that are characterized by

806
00:55:16.599 --> 00:55:22.920
a continuing regional pain that is seemingly
disproportionate in time or degree to the usual

807
00:55:23.000 --> 00:55:29.159
course of any known trauma or other
lesion. That's the official definition. So

808
00:55:29.199 --> 00:55:32.760
basically, these patients have this prolonged
pain. It's completely disproportionate to the initiating

809
00:55:32.800 --> 00:55:37.800
event. Most frequently this is going
to follow a bone or soft tissue injury.

810
00:55:37.079 --> 00:55:40.079
So most cases there's going to be
some sort of injury sprain, fracture,

811
00:55:40.079 --> 00:55:45.039
et cetera. In some cases there
may be no precipitating factor. It's

812
00:55:45.039 --> 00:55:49.079
not common, and then weeks later
they're going to start to develop this range

813
00:55:49.079 --> 00:55:52.840
of clinical manifestations, autonomic dysfunction,
pain out of proportion to the initial injury,

814
00:55:53.119 --> 00:55:57.119
hair and nail changes. The path
though, is really unknown, and

815
00:55:57.119 --> 00:56:00.639
there's some proposed mechanisms, but definitely
nothing to know for the exam. Now,

816
00:56:00.679 --> 00:56:04.880
in clinical manifestations, this is the
most important thing about complex regional pain

817
00:56:04.960 --> 00:56:07.880
syndrome. It's the presentation. The
diagnostic criteria is low yield, the treatments

818
00:56:07.880 --> 00:56:12.960
low yield. It's all about being
able to recognize the clinical manifestations. That's

819
00:56:13.000 --> 00:56:15.559
what you need to know for the
exam. So the main clinical manifestations of

820
00:56:15.599 --> 00:56:22.440
complex regional pain syndrome are pain,
sensory changes, motor impairments, autonomic syntoms

821
00:56:22.480 --> 00:56:25.360
and trophic changes in the affected limb. This is usually going to occur four

822
00:56:25.400 --> 00:56:30.440
to six weeks after the inciting event. The reason I'm covering this under the

823
00:56:30.480 --> 00:56:34.239
upper extremity section for the pants is
because this most commonly occurs in the upper

824
00:56:34.280 --> 00:56:37.599
extremity around sixty percent of the time. So the way that I used to

825
00:56:37.639 --> 00:56:42.119
remember the common clinical manifestations that you're
going to see in a vignette for complex

826
00:56:42.199 --> 00:56:46.199
regional pain syndrome is by remembering instead
of complex regional pain syndrome, I want

827
00:56:46.199 --> 00:56:52.840
you to remember complex regional paint syndrome. So PAI nt complex regional paint syndrome.

828
00:56:53.079 --> 00:56:57.760
And what does paint stand for?
So paint stands for the P stands

829
00:56:57.760 --> 00:57:00.360
for perspiration, and this is due
to the auto aconomic dysfunction, so forty

830
00:57:00.360 --> 00:57:05.960
percent of patients are going to experience
increased sweating on the side where they're experiencing

831
00:57:06.000 --> 00:57:09.800
this. The A in paint stands
for after injury, So after injury for

832
00:57:09.840 --> 00:57:14.760
the A, because remember this is
most commonly going to take place after some

833
00:57:14.800 --> 00:57:16.960
sort of bone or soft tissue injury, So look for some sort of injury

834
00:57:16.960 --> 00:57:22.840
mentioned in the vignette weeks prior the
eye, and PAINT stands for inappropriate pain

835
00:57:22.199 --> 00:57:27.400
because remember the pain experience is inappropriate, it's out of proportion to the initial

836
00:57:27.440 --> 00:57:31.760
injury. Pain is typically the most
prominent in debilitating symptom of complex regional pain

837
00:57:31.800 --> 00:57:37.360
syntems. So remember I stands for
inappropriate pain because it's not appropriate to have

838
00:57:37.360 --> 00:57:38.599
ten out of ten pain in your
risk from a sprain you had two months

839
00:57:38.639 --> 00:57:44.360
ago, So remember inappropriate pain for
I. The END stands for nail changes.

840
00:57:44.559 --> 00:57:47.480
So remember I talked about before your
trophic changes, so these patients may

841
00:57:47.519 --> 00:57:52.559
have increased or decreased nail growth.
Also look for hair growth changes as well,

842
00:57:52.559 --> 00:57:55.159
but remember END stands for nail changes
from your trophic changes. And then

843
00:57:55.199 --> 00:58:00.719
the last letter T and paint stands
for temperature changes. This relates back to

844
00:58:00.760 --> 00:58:04.400
those autonomic changes. Again, these
patients can have and some patients are going

845
00:58:04.440 --> 00:58:07.760
to see a difference in skin temperature
and the affected side versus the unaffected side

846
00:58:08.159 --> 00:58:13.199
of over one degree celsius. So
remember if you have a vignette you think

847
00:58:13.239 --> 00:58:17.519
the patient they're talking about may have
complex regional pain syndrome. Remember to look

848
00:58:17.519 --> 00:58:23.920
for paint perspiration after injury and appropriate
pain, nail changes, temperature, changes,

849
00:58:24.119 --> 00:58:27.519
and that should be enough to get
you there and get the right answer.

850
00:58:27.800 --> 00:58:31.960
Now, diagnosis clinical features based on
your H ANDP, So the diagnosis

851
00:58:32.000 --> 00:58:37.159
is really just based upon the clinical
features determined by your history and physical So

852
00:58:37.360 --> 00:58:39.840
nothing really to know for the exam
question. You're basically looking to see are

853
00:58:39.880 --> 00:58:45.039
they having pain, sensory changes,
motor symptoms, autonomic dysfunction? Are these

854
00:58:45.039 --> 00:58:49.559
symptoms weeks out from an initial injury
that would no longer be appropriate to persist

855
00:58:49.599 --> 00:58:52.880
at this point in time. There
are some imaging tests that can be used

856
00:58:52.960 --> 00:58:57.039
three phase bone syntigraphy, radiographs,
but there's nothing high yield, there's no

857
00:58:57.119 --> 00:59:00.400
gold standard tests. Definitely nothing I
would memorize for the exam, So just

858
00:59:00.440 --> 00:59:05.000
know this is generally clinical diagnosis.
Are the experiencing paint? If so?

859
00:59:05.239 --> 00:59:13.559
Complex regional pain syndrome treatment it's multifaceted
physical therapy and SAIDs tricyclic antidepressence, sympathetic

860
00:59:13.599 --> 00:59:17.800
nerve blocks. Physical and occupational therapy
are more or less considered first line treatment

861
00:59:19.159 --> 00:59:23.440
for complex regional pain syndrome. But
again it's a multifaceted, multi disciplinary approach.

862
00:59:23.480 --> 00:59:27.920
There's not one specific thing to know
for treatment. The main takeaway for

863
00:59:28.000 --> 00:59:35.280
complex regional pain syndrome. To identify
in a vignette is to remember the symptoms

864
00:59:35.320 --> 00:59:37.840
that you're seeing in these patients.
The treatments really not high yield. Diagnostic

865
00:59:37.840 --> 00:59:42.079
criteria is really not that high yield. It's being able to recognize it in

866
00:59:42.119 --> 00:59:46.159
a vignette. So remember complex regional
paint syndrome, perspiration after injury, inappropriate

867
00:59:46.239 --> 00:59:50.639
pain, nail changes, temperature changes. That's the main takeaway here, and

868
00:59:50.760 --> 00:59:53.199
we are done with the upper extremity. Let's do five quick questions. Question

869
00:59:53.239 --> 00:59:57.920
one, thirty two year old female
presents the office complaining of pain at the

870
00:59:58.039 --> 01:00:01.119
radial sign of her wrist that is
most prominent upon movement of the thumb.

871
01:00:01.400 --> 01:00:07.039
She has four weeks postpartum. Pasthematical
history is otherwise unremarkable. On exam,

872
01:00:07.039 --> 01:00:10.800
tenderness is noted over the radial styloid
and pain. His experience with passive ulner

873
01:00:10.840 --> 01:00:15.599
deviation of the wrist with the thumb
flexed in the palm. What is the

874
01:00:15.679 --> 01:00:20.039
likely diagnosis in this patient? So
that is going to be decore vain tendinopathy.

875
01:00:20.400 --> 01:00:23.800
So decarevyin tendonopathy we know is most
commonly seen in women between the thirty

876
01:00:23.840 --> 01:00:29.199
to fifty year age rage also common
to see and postpartum women four to six

877
01:00:29.239 --> 01:00:32.079
weeks after delivery. So she fits
this criteria to the tea. Then she

878
01:00:32.119 --> 01:00:35.559
states she has pained on the radio
side of the wrist, and then a

879
01:00:35.559 --> 01:00:38.639
physical exam tenderness over the radio styloid. She has a positive Finkelstein test for

880
01:00:38.760 --> 01:00:44.119
the patient's wrist as put into Olnar
deviation. All the patient's fingers are folded

881
01:00:44.159 --> 01:00:47.800
over the thumb. This is a
classic presentation for decorevain tendeonopathy. Question two.

882
01:00:47.880 --> 01:00:52.199
A forty three year old female presents
at the office complaining of numbness and

883
01:00:52.320 --> 01:00:55.960
tingling in her hands, mostly affecting
the thumb, index and middle finger,

884
01:00:57.199 --> 01:01:00.519
and part of the ring finger.
She states it is worse at nights,

885
01:01:00.559 --> 01:01:04.400
sometimes awaking her from sleep. Both
a Tannell and a Palin test are positive.

886
01:01:04.400 --> 01:01:08.119
On physical exam. His patient is
likely experiencing compression of which nerve,

887
01:01:08.480 --> 01:01:12.559
so that is going to be the
median nerve. So this patient is experiencing

888
01:01:12.599 --> 01:01:15.480
carpal tunnel syndrome, which is compression
of the medial nerve median nerves. So

889
01:01:15.519 --> 01:01:20.280
we know this because the patient parastages
in the median nerve. Territory, which

890
01:01:20.320 --> 01:01:22.920
will be the first three fingers and
radial half of the fourth. In addition,

891
01:01:22.960 --> 01:01:27.239
she states it's worse at night,
which is very common for carpal tunnel.

892
01:01:27.559 --> 01:01:30.039
Finally, we have a positive Tannell
and Phalin test that seals the deal.

893
01:01:30.280 --> 01:01:34.679
We know this is carpal tunnel,
which is median nerve compression. Question

894
01:01:34.760 --> 01:01:37.639
three. Twenty seven year old Mail
presents to the er after a bicycle accident

895
01:01:37.679 --> 01:01:40.840
he had earlier on in the day. He states his bike hit a pothole

896
01:01:40.880 --> 01:01:45.039
which sent him flying off his bike, landing onto his outstretched hands. He's

897
01:01:45.079 --> 01:01:50.280
now complaining of pain along the radial
side of the wrist and is tender just

898
01:01:50.440 --> 01:01:55.039
proximal to the base of the thumb
at the anatomic snuffbox fracture of which bone

899
01:01:55.280 --> 01:01:59.920
should be suspected. And this patient
until proven otherwise, so that is going

900
01:02:00.000 --> 01:02:02.599
to be the scaffoid. This is
a simple one scaffoid or avicular fracture.

901
01:02:02.639 --> 01:02:06.519
You have a patient had to fall
into an outstretched hand, which is often

902
01:02:06.559 --> 01:02:08.239
the mechanism of injury for a scaffoid
fracture, and he has pain on the

903
01:02:08.320 --> 01:02:13.119
radial side of the wrist. Snuffbox
tenderness. You are done. That is

904
01:02:13.119 --> 01:02:16.079
a scaffoid fracture until proven Otherwise,
as soon as you hear snuffbox, always

905
01:02:16.079 --> 01:02:21.519
be thinking of a scaffoid fracture.
Question four. Sixty seven year old Mail

906
01:02:21.559 --> 01:02:24.559
presents to the office today complaining of
persistent elbow pain. He does not recall

907
01:02:24.599 --> 01:02:29.039
any trauma to the elbow, but
the painting he is experiencing in his elbow

908
01:02:29.119 --> 01:02:31.639
is affecting his golf game, as
he is an avid golfer. On exam,

909
01:02:31.679 --> 01:02:37.280
pain is elicited by performing risk flection
against resistance. Tenderness would likely be

910
01:02:37.320 --> 01:02:42.360
felt in which part of the elbow
in this patient, so that would be

911
01:02:42.400 --> 01:02:46.039
at the medial epicondyle. So you
have a classic case of medial epicondolitis aka

912
01:02:46.159 --> 01:02:50.679
golfer's elbow. If you have a
sixty seven year old male avid golfer with

913
01:02:50.800 --> 01:02:53.239
elbow pain, no preceding trauma,
and then the key is that the pain

914
01:02:53.360 --> 01:02:58.440
is reproduced on exam with the risk
being flexed against resistance. So in this

915
01:02:58.480 --> 01:03:01.360
case the patient wouldn't have pain and
the medial epicondyle. And then of course

916
01:03:01.360 --> 01:03:05.679
remember the demonic mini golf is fun
and in mini helps you Remember this is

917
01:03:05.679 --> 01:03:09.599
the media up a condal involved in
golfer golfer's elbow golfs because this is known

918
01:03:09.679 --> 01:03:13.559
as golfer's elbow. And then the
f and fun helps you remember this is

919
01:03:13.559 --> 01:03:17.039
going to involve flection, whether it's
pain as pain with risk, flection against

920
01:03:17.039 --> 01:03:22.239
resistance on exam the fact that it
involves the flex or copper radialis or that

921
01:03:22.280 --> 01:03:25.119
it's caused from repetitive flection. Mini
golf is fun helps you remember all the

922
01:03:25.159 --> 01:03:28.719
things that you need to know.
And then by method of exclusion, you

923
01:03:28.760 --> 01:03:32.320
know lateral epicond of lytis is the
opposite problems with extension not flection. Last

924
01:03:32.400 --> 01:03:37.400
question question five. Seventeen year old
Mail presents to the er after sustaining an

925
01:03:37.400 --> 01:03:39.920
injury to his right arm. After
X rays are complete, the attending physician

926
01:03:39.960 --> 01:03:45.519
assistant and forbs him that the X
ray revealed a proximal owner fracture accompanied by

927
01:03:45.559 --> 01:03:52.679
a radial head dislocation. This type
of injury is also known as a and

928
01:03:52.719 --> 01:03:57.440
that is going to be a Montegia
fracture. So remember grew some murder Montegia

929
01:03:57.519 --> 01:04:01.400
and Galiazzi fracture grew some first lead
letters in russom stand for Galiazzi our stands

930
01:04:01.400 --> 01:04:05.960
for radius fracture, U stands for
owning dislocation aka the radio owner joint,

931
01:04:06.039 --> 01:04:11.920
and then murder, which is the
first three letters mu R montigia U stands

932
01:04:11.920 --> 01:04:15.719
for owner fracture, and our stands
for radiohead dislocation, which is the type

933
01:04:15.719 --> 01:04:17.480
of injury we see here in this
patient. All right, that is the

934
01:04:17.559 --> 01:04:20.800
upper extremity for the MSK section for
the pants. Thank you so much for

935
01:04:20.840 --> 01:04:25.400
listening to the podcast, and good
luck on you in paschool. Good luck

936
01:04:25.400 --> 01:04:27.760
on your pants or pantry ears,
and thank you again

